Abstract
Myasthenia gravis is an organ-specific autoimmune disease characterized by the production of anti-acetylcholine receptor antibodies. In this review, I describe the pathophysiological importance of the altered chemokine receptor-mediated signaling in the thymus and peripheral blood of myasthenia gravis patients. The epidemiological and clinical features of myasthenia gravis are also discussed.
MeSH terms
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Animals
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Autoantibodies / immunology
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Autoimmune Diseases / epidemiology
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Autoimmune Diseases / etiology
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Autoimmune Diseases / immunology
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Autoimmune Diseases / physiopathology
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Chemokines / immunology*
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Humans
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Models, Biological
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Myasthenia Gravis / epidemiology
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Myasthenia Gravis / etiology*
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Myasthenia Gravis / physiopathology
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Receptors, Chemokine / immunology
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Thymus Gland / immunology*
Substances
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Autoantibodies
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Chemokines
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Receptors, Chemokine