Article Figures & Data
Figures
- Figure 1
- An axial FLAIR image illustrates A-B) 2 hyperintense lesions in the cerebellar peduncles along with a lesion in the splenium of the corpus callosum. C-D) illustrate an axial T2 image of the same lesions, and E) demonstrates a sagittal T1 image which shows the clear involvement of the splenium along with a generalized atrophy of the corpus callosum. These MRI are consistent with Marchiafava-Bignami disease (MBD).
- Figure 2
- Illustrates the resolution of the identified lesions upon presentation.
- Figure 3
- Illustrates the timeline of our patient’s presentation.
Tables
Yıldırım Z et al8 Bachar M et al9 Salazar G et al10 Alshimemeri S et al (our paper) Demographics 58yo, F 44yo, F 62yo, F 45yo, F Type of bariatric surgery Intragastric balloon Roux-en-Y gastric bypass Not specified Laparoscopic sleeve gastrectomy Other comorbidities None Schizophrenia and bipolar disorder. None None Interval between surgery and presentation 7 days 23 years 5 years 40 days Laboratory findings Hypokalemia High MCV, low albumin, elevated LFTs, high ammonia levels. Low vitamin B12 and B9 levels Hypernatremia, elevated LFTs and high glucose and protein in her CSF Lesions’ location Splenium of the CC Splenium of the CC and posterior limb of internal capsule The entire corpus callosum. Splenium of the CC and cerebellar peduncles Treatment IV fluids, IV K+, metoclopramide, Vitamin supplementation, antipsychotics. Vitamin replacement, MgSO4, antipsychotics, corticosteroids. Vitamin replacement, Anti-Epileptics. IV fluids, Vitamin replacement, Diphenhydramine, and metoclopramide Recovery Complete Complete Substantial recovery, however incomplete Substantial recovery, however incomplete Follow-up period 14 days 10 days 3 months 6 months F: Female, MCV: Mean cell volume, LFTs: Liver function tests, CSF: Cerebrospinal fluid, CC: Corpus callosum.
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